Haem-activated promiscuous targeting of artemisinin in Plasmodium falciparum

A Dynamic Stress Model Explains the Delayed Drug Effect in Artemisinin Treatment of Plasmodium falciparum

Mechanisms of artemisinin resistance in Plasmodium falciparum malaria

Frontiers A Malaria Parasite Cross Reveals Genetic Determinants of Plasmodium falciparum Growth in Different Culture Media

Plasmodium falciparum: multifaceted resistance to artemisinins, Malaria Journal

High-level artemisinin-resistance with quinine co-resistance emerges in P. falciparum malaria under in vivo artesunate pressure, BMC Medicine

High-level artemisinin-resistance with quinine co-resistance emerges in P. falciparum malaria under in vivo artesunate pressure, BMC Medicine

Frontiers Artemisinin: A Panacea Eligible for Unrestrictive Use?

Mechanisms of artemisinin resistance in Plasmodium falciparum malaria - ScienceDirect

A Dynamic Stress Model Explains the Delayed Drug Effect in Artemisinin Treatment of Plasmodium falciparum

Protein abundance and folding rather than the redox state of Kelch13 determine the artemisinin susceptibility of Plasmodium falciparum - ScienceDirect

PDF] Chemical proteomics approach reveals the direct targets and the heme-dependent activation mechanism of artemisinin in Plasmodium falciparum using an artemisinin-based activity probe

Peroxide antimalarial drugs target redox homeostasis in Plasmodium falciparum infected red blood cells

PfHDAC1 is an essential regulator of parasite asexual growth with its altered genomic occupancy and activity associated with artemisinin drug resistance in Plasmodium falciparum